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In the News
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TREATMENT OF DIABETIC FOOT WOUNDS
Diabetic foot wounds are complex microcosms of multiple pathophysiologic processes. The wounds are predominantly characterized by polymicrobial infection, peripheral neuropathy, structural deformity, altered immune function or increased susceptibility to infection, decreased wound nitric oxide production, and often hypoxia/ischemia. Decreased NO production, infection, and hypoxia are the most important for inhibition of wound healing. Decreased NO production is responsible for impaired cutaneous vasodilation, decreased neurogenic vascular response, diabetic neuropathy, and endothelial cell dysfunction that inhibit the processes necessary for granulation tissue formation.
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- IHMA Cover Letter
"It would seem plausible that if in our hospitals severe surgery would be followed by the use of Hyperbaric Oxygen, healing would be accelerated by 30%. This alone would result in the saving of billions of dollars, not to even mention the reduction in human suffering."
- THE APPLICATION OF NFPA HYPERBARIC FACILITY RULES
The National Fire Protection Association (NFPA) document NFPA 99, Standard for Healthcare Facilities, is the primary safety standard addressing the safety of medical gas and electrical services in US healthcare facilities. Its Chapter 19 "Hyperbaric Facilities contains a comprehensive set of safety requirements for hyperbaric chambers used in healthcare settings. Further, NFPA 99 rules for hyperbaric facilities are generally not voluntary.
- Whole Brain Radiation and Solitary Brain Metastasis
The initial approach to using radiation postoperatively to treat brain metastases, used to be whole brain radiation, but this was abandoned because of the substantial neurological deficits that resulted, sometimes appearing a considerable time after treatment. Whole brain radiation was routinely administered to patients after craniotomy for excision of a cerebral metastasis in an attempt to destroy any residual cancer cells at the surgical site. However, the deleterious effects of whole brain radiation, such as dementia and other irreversible neurotoxicities, became evident.
- HBOT in the Treatment of Chronic Brain Injury
May 2, 2002 Case Study using Hyperbaric Oxygen Treatment and SPECT imaging for analysis.
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- Evidence for a restorative effect of low pressure HBOT on chronic brain injury
On May 2, 2002 Dr. Paul G. Harch, Clinical Assistant Professor of Medicine of the Louisiana State University School of Medicine, New Orleans was invited and presented evidence for a restorative effect of low pressure HBOT on chronic brain injury before the Subcommittee on Labor, Health, Human Services, and Education of the House of Representatives Appropriations Committee. The testimony consisted of functional brain imaging (SPECT) documentation of improvements in brain blood flow in 15 patients with a variety of chronic brain injuries.
- Hyperbaric Medicine Officially Enters the Prohibition Era
The Physicians' Forum article in the inaugural issue is a comprehensive discourse with many good points on the legality of off-label HBOT, research funding, a registry, recommendations for patients seeking off-label treatment, and standardized testing/documentation. However, these points are nearly lost in an extremely confusing, obfuscating tangle of terms, definitions, and concepts that is partly based on a double standard.
- The Dosage of Hyperbaric Oxygen in Chronic Brain Injury
The concept of dosage of hyperbaric oxygen therapy (HBOT) derives from the definition of HBOT as a drug. Using the broad definition of HBOT by Harch and Neubauer (1), HBOT is the use of greater than ambient pressure oxygen as a drug to treat basic pathophysiologic processes/states and their diseases. Drug dosage of HBOT, therefore, is a function of baseline or reference ambient pressure, depth of pressurization, duration, frequency, air breaks, surface interval, number of treatments, idiosyncratic genetic patient factors, and time to intervention in the disease process which determines the pathological targets
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