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Controversies in the treatment of acute carbon monoxide intoxication

Acute carbon monoxide (CO) poisoning causes brain lipid peroxidation as well as an hypoxic injury. Traditional therapy has been 1 ATA oxygen but now includes hyperbaric oxygen therapy (HBOT) which hastens dissociation of COHgb, reverses anoxia, inhibits reperfusion injury, and has toxic effects. All of these are dose dependant and time sensitive. The human experience with HBOT is anecdotal cases, uncontrolled and controlled series, which are largely positive, and randomized, prospective e controlled studies with conflicting results. A controversy exists over the pressure and number of HBOTs.

HMPAO SPECT Brain Imaging of Acute CO Poisoning and Delayed Neuropsychological Sequelae (DNSS)

PG Harch, KW Van Meter, SF Gottlieb, P Staab. JoEllen Smith Hyperbaric Medicine Unit, New Orleans, LA 70131

Background: Brain CT and MRI are poor barometers of acute CI poisoning and the DNSS syndrome, registering abnormalities in a minority of patients. HMPAO SPECT, a flow metabolism modality, seems more appropriate. We report 12 cases of acute poisoning and DNSS imaged with HMPAO SPECT/

Hyperbaric Oxygen Therapy Treatment of Neurological Residual of Carbon Monoxide Poisoning

Paul G. Harch, Keith Van Meter, Paul K Staab, Sheldon F. Gottlieb, Jo Ellen Smith Medical Center, New Orleans, LA.

There is no accepted treatment for neurological residual of carbon monoxide poisoning; this report documents the effect of HBOT for established neurological residual.

Pediatric Cerebral Palsy treated by 1.5 ATA hyperbaric oxygen therapy, a pilot study

Kevin Barrett, M.D., F.A.C.P. Ð Professor of Hyperbaric Medicine
University of Texas Medical Branch, Galveston, TX, USA


Authors: Kevin F. Barrett M.D., Kevan P. Corson CHt, Jon T. Mader, M.D.

Title: Pediatric Cerebral Palsy treated by 1.5 ATA hyperbaric oxygen therapy, a pilot study.

Objective: To determine if 1.5 ATA hyperbaric oxygen therapy can ameliorate the neurologic deficits associated with pediatric cerebral palsy.

Low-Pressure Hyperbaric Oxygen Therapy (HBOT) as an Investigational Treatment for Developmental Disabilities

Low-Pressure Hyperbaric Oxygen Therapy (HBOT) as an Investigational Treatment for Developmental Disabilities

Hyperbaric oxygen therapy (HBOT) involves the inhalation of 100% oxygen inside a chamber pressurized above sea-level atmospheric pressure. HBOT substantially increases the amount of oxygen dissolved in all bodily fluids, including blood plasma, cerebrospinal fluid, and lymph. Increased oxygen in these fluids provides oxygen directly to underoxygenated (hypoxic) tissues, supplementing the usual transport system by hemoglobin molecules in the blood.

HBOT adjunctive role in the treatment of Autism

Hyperbaric Oxygen Therapy (HBOT) adjunctive role in the treatment of Autism

Trish Planck
Clinical Director of Hyperbarics
Hyperbaric Oxygen Clinic of Nevada
Hyperbaric Oxygen Clinic of Santa Monica

Autism is a complex neurological disorder that typically occurs within the first three years of life that results in usually severe developmental disability. Its incidence in the population is estimated to be 1 in 500 with a prevalence of boys to girls of 4 to 1. The ensuing effects of the disease result in severe impairment in areas of social interaction and communication and in some individuals self-injurious behavior may occur.

Hyperbaric Oxygenation Therapy in Patients with Chronic Childhood Encephalopathy

Solany Zervini, M.D. Chief of Hyperbaric Medicine, Centro Medico

Hiperbario do Rio de Janeiro

Chronic childhood encephalopathy (CP) remains a shadow in neuropediatrics. No treatments are proposed except physiotherapy, occupational therapy and phonoaudiology which may last for years at times and as long as the patient lives. None of these therapies bring enough benefits and the prognosis of the disease is still very poor.

Generic inhibitory drug effect of hyperbaric oxygen therapy (HBOT) on reperfusion injury (RI)

P.G. Harch

LSU School of Medicine, New Orleans, LA, USA

Introduction HBOT is the use of high pressure 02 as a drug to treat pathophysiologic processes and their diseases. The purpose of this paper is to see if HBOT has a generic inhibitory drug effect on RI.

Methods Review of animal studies on HBOT in RI.



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