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LSU, Miles seeking solutions to the concussion problem

August 12, 2013-© 2013 Tiger Rag [Re-Post permission acquired]

University teaming up with New Orleans doctors who believe they have found the answer

University teaming up with New Orleans doctors who believe they have found the answer

LSU is working with doctors in New Orleans who might just hold the key to eliminating both long and short-term concussion effects. If their research treating brain injuries with hyperbaric oxygen therapy proves fruitful, one of the most pressing issues in sports could finally be showing some light at the end of a long, dark tunnel.


Hyperbaric oxygen therapy may revolutionize the way concussions are treated.
Tiger Rag Assistant Editor

LSU coach Les Miles endorses use of hyperbaric oxygen treatment


the more oxygen you put into your system for a variety of things the better it is,” Miles said recently.

His personal revelation led Miles to start peppering LSU’s medical staff with questions about the potential of oxygen treatment for injuries. Eventually, it led Miles to the discovery that a pair of doctors in New Orleans, Paul Harch and Keith Van Meter, were at the forefront of hyperbaric oxygen therapy...

A Phase I Study of LP HBOT for Blast-Induced TBI, PCS, and PTSD

A Phase I Study of Low-Pressure Hyperbaric Oxygen Therapy for Blast-Induced Post-Concussion Syndrome and
Post-Traumatic Stress Disorder


Traumatic Brain Injury Journal of Neurotrauma

Eight active duty and eight recently retired servicemen were self-referred or referred by their military commanders/physicians. Fourteen subjects had pre-study diagnoses of TBI/PCS with PTSD, and two subjects had TBI/PCS. Prestudy diagnostic evaluations and criteria were not available to the study authors. All subjects underwent brain MRI in the military prior to treatment. All subjects gave informed consent and enrolled in LSU IRB #7051.

Army major seeing success with LSU hyperbaric treatment

Army Major Ben Richards Seeing Success with LSU Hyperbaric Oxygen Treatment


Posted on November 7, 2012 at 6:36 PM

Updated today at 6:41 PM


Meg Farris / Eyewitness News
Email: | Twitter: @megfarriswwl

Good News on Veteran's Day 2012: Treatment for PTSD and TBI Among Veterans Shows Results

Good News on Veteran's Day 2012: Treatment for PTSD and TBI Among Veterans of Iraq and Afghanistan Shows Results

Paul G. Harch, M.D. announces successful treatment of PTSD (Post Traumatic Stress Disorder) and TBI (Traumatic Brain Injury) for veterans suffering from debilitating concussive brain injury.

(New Orleans, LA) November 05, 2012 -- Following publication of the first case of HBOT (Hyperbaric Oxygen Therapy) benefiting veterans with TBI and PTSD in 2009, Dr. Harch and colleagues published confirmatory evidence in January of this year in the prestigious peer-reviewed Journal of Neurotrauma.

The Advocate Baton Rouge Newspaper: "LSU will study brain injury treatments"

LSU will study brain injury treatments

The Advocate February 6, 2009


Dr. Paul Harch, an LSU Health Sciences Center emergency medicine professor, is starting a pilot study on treating people with chronic traumatic brain and post traumatic stress disorder, according to a statement from LSU Health Sciences Center.

The study will examine 30 participants, half with traumatic brain injury and half with traumatic brain injury and post-traumatic stress disorder, the statement says. Participants will undergo oral, written and computer tests as well as brain imaging before and after receiving 40 to 80 treatments of hyperbaric oxygen therapy. Researchers are looking to see if the therapy improves thinking ability, and quality of life and reduces post-traumatic stress disorder symptoms. The U.S. Centers for Disease Control and Prevention estimates 2 percent of the population needs long-term assistance because of a traumatic brain injury, the statement says. Traumatic brain injury has been the “signature” injury of the ongoing wars in Iraq and Afghanistan, the statement says. For more information or to determine if you qualify, call (504) 309-4948.


Leslie Capo
Director of Information Services
LSU Health Sciences Center New Orleans

"The Secret to Growing New Brain Cells: Sleep, Exercise and Hyperbaric Oxygen?" by Veronica Fern-McElarney, Stanford Univ

The Secret to Growing New Brain Cells: Sleep, Exercise and Hyperbaric Oxygen?

Veronica Fern-McElarney


A 33-year-old drunk driver wraps his pick-up truck around a tree and is brought to the emergency room at a small, community hospital in Slidell, Louisiana.   His emergency room doctor, Dr. Paul Harch, recalled the scene.  “You know, high-speed, straight into a pick-up, no seatbelt, and the flexion injury rendered him paralyzed immediately. By the time they got him off the floorboard of the truck…he had a flicker of movement in his one big toe; within forty minutes he was densely paraplegic. At three in the morning the neurosurgeon, radiologist and myself looked at each other…and the only explanation was that he had a vascular injury to his spinal cord. And almost in unison we said,  “Gosh, I wonder what he could do with a little oxygen?” I put him in the hyperbaric chamber and he moved his toe. When we took him out, he had sensation down in the foot. Incrementally, every time I put him in, he got more and more sensation. In seventeen days, he walked out of the hospital. That just blew everybody away.”

TBI/PTSD DoD Hearing/Army Times Article/ Score Card 16 out of 16 improved with HBOT

For those wondering about how hyperbaric medicine came up in yesterday's House appropriation hearing, it was because you have a lobbyist in Washington, D.C. There have been all kinds of stories lately about how terrible lobbyists are. Of course, when you have one working for your interests, hopefully that will make a difference.

H-140 is a very small hearing room for DoD hearings. They only permitted 5 members of the public and about 5 press members into the room. I waited outside in the cold for an hour before we were permitted into the Capitol. Because I was there, Chairman Murtha called on me from the audience to talk about the score card and whether the people on it were real people. I let him know that he actually knew 3 of the 16 people. He knew the Brigadier General who was the first patient treated, one of the physicians responsible for 3 of the Airmen is actually the father of a Marine Hero from the Iraq war, and he knows the Navy SEAL who was returned to duty as well.

The Dosage of Hyperbaric Oxygen in Chronic Brain Injury

The Dosage of Hyperbaric Oxygen in Chronic Brain Injury

Paul G. Harch M.D.


The concept of dosage of hyperbaric oxygen therapy (HBOT) derives from the definition of HBOT as a drug. Using the broad definition of HBOT by Harch and Neubauer (1), HBOT is the use of greater than ambient pressure oxygen as a drug to treat basic pathophysiologic processes/states and their diseases. Drug dosage of HBOT, therefore, is a function of baseline or reference ambient pressure, depth of pressurization, duration, frequency, air breaks, surface interval, number of treatments, idiosyncratic genetic patient factors, and time to intervention in the disease process which determines the pathological targets. All of these factors cause HBOT to be a narrow-window drug in chronic brain injury similar to digoxin and coumadin: too litfie maybe ineffective and too much can be toxic. In addition, oxygen is a respiratory metabolite: ;too little has serious metabolic consequences and too much can cause metabolic fatigue. Determining the proper dosage in a given patient with a specific or multiple diseases can be difficult. Ultimately, one wants the best dosage that improves the patient while doing the least harm.

HBOT has both acute and chronic effects (2). This paper will address only the chronic effects. Chronic effects of HBOT include fibroblast stimulation, collagen deposition, anglogenesis, epithelialization, and bone remodeling. This is most evident in shallow perfusion gradient wounds such as the classic homogenous wound of external beam radiation. In this animal and human model, Marx (3) has shown that intermittent exposure to HBOT induces the aforementioned chronic trophic effects to cause wound healing. The final level of tissue oxygenation after HBOT is approximately 80% of normal tissue, but the effect is durable for years. The unproven mechanism of the effect is thought to be secondary to transient elevation of tissue oxygen levels that results in a steep oxygen gradient that causes anglogenesis. Since 1995 this effect has been better characterized as signal induction where the drug HBOT, by elevation of tissue oxygen pressures, alone or in combination with other factors, signals the DNA to begin transcription of various gene sequences to mRNA (4,5,6). The mRNA is translated to proteins which cause trophic tissue changes, i.e., wound healing. These mechanisms are thought to be responsible for the HBOT-induced wound healing that is seen in a large variety of chronic non-healing wounds, such as arterial insufficiency, venous insufficiency, diabetic, radiation, sickle cell, vasculitic, and other ulcers.


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