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Last Updated: April 4 , 2005 9:32 AM

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Silicone Adjunctive Disease

Beginning the 12th of August 1998 the day of her explantation surgery, Sally has been bringing media attention (television, radio, and print) to silicone adjunctive disease. Her support group will actively promote the awareness and support for the thousands of individuals detrimentally effected by implants (of silicone, saline, or other chemicals). Sally's personal illness/health-struggle has been epic and she has opened her life as an example of survival to others who are suffering from the negative effects of implants. She is researching, with other women, support groups and top medical and holistic doctors. more >

In the News 
Top Stories
Chronic Fatigue Syndrome Articles
In a subset of chronic fatigue syndrome (CFS) patients, the immune system is always activated. Although it is unknown why this happens, one hypothesis is that it is caused by a lingering infection or an infection that leaves an autoimmune sequelae.
Hyperbaric Oxygen Therapy is proven effective in the treatment of Cerebral Palsy
The recent Quebec, Canada study on the effectiveness of hyperbaric oxygen as a treatment modality for children with cerebral palsy, reported in the prestigious British medical journal, The Lancet (2/24/01), has reaffirmed the positive effects of hyperbaric therapy," concludes J. Michael Uszler, M.D., internationally recognized expert on functional brain imaging


More News Items
  • FEAT DAILY ONLINE NEWSLETTER
    In a disturbing and saddening update, the latest figures from the California Department of Developmental Services of new children diagnosed with autism reveals a new, rapid rate of increase across the state. The new figures covers a 36 month period, from April of 1996 to as recently as April of this year.
  • Brain SPECT – Relationship to HBOT
    In the last few years Hyperbaric Oxygen Therapy (HBOTx) has begun to have a new therapeutic endpoint – the improvement of long-term brain disorders. Brain SPECT imaging has begun to be utilized in evaluating the effects of that therapy.
    March 11, 2003, 8:20 PM PST
  • Investigational Treatment for Developmental Disabilities
    Hyperbaric oxygen therapy (HBOT) involves the inhalation of 100% oxygen inside a chamber pressurized above sea-level atmospheric pressure. HBOT substantially increases the amount of oxygen dissolved in all bodily fluids, including blood plasma, cerebrospinal fluid, and lymph. Increased oxygen in these fluids provides oxygen directly to underoxygenated (hypoxic) tissues, supplementing the usual transport system by hemoglobin molecules in the blood
  • ETHICS OF DOUBLE BLIND STUDIES IN CHILDREN
    Doing basic research on children poses an extremely emotional problem. It is a great consternation to the mothers to know that one child who will receive air, the other child will receive hyperbaric oxygen. This is especially true when there is a known risk factor at the time of the delivery such as cord strangulation, stroke and some type of severe hypoxic ischemic episode that may be benefited by immediate hyperbaric oxygenation.
  • HMPAO SPECT Brain Imaging of Acute CO Poisoning and Delayed Neuropsychological Sequelae
    Brain CT and MRI are poor barometers of acute CI poisoning and the DNSS syndrome, registering abnormalities in a minority of patients. HMPAO SPECT, a flow metabolism modality, seems more appropriate. We report 12 cases of acute poisoning and DNSS imaged with HMPAO SPECT/
  • LOW PRESSURE HYPERBARIC OXYGEN THERAPY ( LPHBOT ) INDUCES CEREBROVASCULAR CHANGES AND IMPROVES COGNITIVE FUNCTION IN A RAT TRAUMATIC BRAIN INJURY
    In 1996 pilot data were reported on this project [UHM, 1996, 23: (Suppl); 48]. The present study ia a replication of the cognitive improvements and blood vessel changes in that experiment using a larger number of rats
  • The Effect of HBOT Tailing Treatment on Neurological Residual and PSCT Brain Images in Type II
    No guidelines exist for HBOT tailing treatment of residual neurological injury and no studies document their effect on SPECT brain images and neurological condition in Type II/CAGE DCI as we report herein.
  • Chronic Childhood Encephalopath
    Chronic childhood encephalopathy (CP) remains a shadow in neuropediatrics. No treatments are proposed except physiotherapy, occupational therapy and phonoaudiology which may last for years at times and as long as the patient lives. None of these therapies bring enough benefits and the prognosis of the disease is still very poor
  • HBOT) adjunctive role in the treatment of Autism
    Autism is a complex neurological disorder that typically occurs within the first three years of life that results in usually severe developmental disability. Its incidence in the population is estimated to be 1 in 500 with a prevalence of boys to girls of 4 to 1. The ensuing effects of the disease result in severe impairment in areas of social interaction and communication and in some individuals self-injurious behavior may occur.
  • HYPERBARIC OXYGEN THERAPY FOR CEREBRAL PALSY
    From 1992 to 1999, 26 children with CP or static encephalopathy were evaluated under a unique neuororehabilitation program of the Baromedical
  • Kevin Barrett, M.D., F.A.C.P. – Professor of Hyperbaric Medicine
    Five children, average age 41.8 months, were treated with 1.5 ATA hyperbaric oxygen therapy (HBOT) for a total of sixty treatments administered for one hour daily, five days a week. A modified test of gross motor and fine motor function (GMFM-m) and a modified Ashworth Spasticity Scale were employed before and after hyperbaric therapy.
  • TREATMENT OF NEUROLOGICAL RESIDUAL OF CARBON MONOXIDE POISONING
    5 adult males (29-51 y.o.) presented 6-10 months after CO poisoning. 4 were group poisoned (1 episode), the 5th was poisoned dozens of times over 3.5 years. All had cognitive, neurological, and constitutional symptoms with primarily balance, gait, and coordination abnormalities on physical exam.
  • Controversies in the treatment of acute carbon monoxide intoxication
    Acute carbon monoxide (CO) poisoning causes brain lipid peroxidation as well as an hypoxic injury. Traditional therapy has been 1 ATA oxygen but now includes hyperbaric oxygen therapy (HBOT) which hastens dissociation of COHgb, reverses anoxia, inhibits reperfusion injury, and has toxic effects. All of these are dose dependant and time sensitive.
  • Cognitive and Cerebral Blood Flow Improvement in Chronic Stable Traumatic Brain Injury Induced by 1.5 ATA Hyperbaric Oxygen.
    Following severe traumatic brain injury, cognitive improvement is most dramatic the first six months following injury and largely static after 18 months. Anecdotal reports exist that attest to the efficacy of HBOT to improve posttraumatic neurologic deficits by increasing blood flow in the ischemic penumbra despite protocol differences, CBF, speech, neurological and cognitive testing have not been studied serially in patients undergoing HBOT for chronic stable TBI.
  • Delayed Treatment of Type II DCS: The Importance of Low Pressure HBOT and HMPAO SPECT Brain Imaging in its Diagnosis and Treatment.
    We describe a prospective case study of a sport SCUBA diver with Type II DCS (brain) in which HMPAO SPECT brain imaging with oxygen intervention was used in the identification of potentially recoverable brain tissue which subsequently responded to low pressure HOBT as demonstrated by improvement in neurological functions and SPECT brain images.






Brain Injury

Cerebral Blood Flow Changes and Cognitive Improvement in Chronic Stable Traumatic Brain Injuries

Pharmacology and Effects on Pathophysiology in Acute Central Nervous System Ischemia and Anoxia

Hyperbaric Oxygen Therapy in Global Cerebral Ischemia/ Anoxia and Coma

Hyperbaric Oxygen therapy in global cerebral ischemia/anoxia and coma

HOTFAST: Hyperbaric Oxygen Therapy for Acute Stroke

Cerebral Palsy

YPERBARICOXYGEN THERAPY FOR CEREBRAL PALSY AND STATIC ENCEPHALOPATHY

Hyperbaric Oxygen Therapy in the Treatment of Spastic Diplegia Cerebral Palsy

Hyperbaric Oxygen Therapy for Cerebral Palsy and Static Encephalopathy of Childhood

Imaging

Generic inhibitory drug effect of hyperbaric oxygen therapy (HBOT) on reperfusion injury

THE IMPORTANCE OF LOW PRESSURE HBOT AND HMPAO SPECT BRAIN IMAGING IN ITS DIAGNOSIS AND TREATMENT.

HMPAO SPECT BRAIN IMAGING AND LOW PRESSURE HBOT IN THE DIAGNOSIS AND TREATMENT OF CHRONIC TRAUMATIC, ISCHEMIC, HYPOXIC AND ANOXIC ENCHPHALOPATHIES.

Wound Healing

MOLECULAR BIOCHEMISTS UNLOCKING FUNDAMENTAL MECHANISMS OF HYPERBARIC OXYGEN THERAPY IN WOUND HEALING AND REPERFUSION INJURY

UHMS

PRO ARGUMENTFOR INCLUSION OF ACUTE TRAUMATIC BRAIN INJURYON THE UHMS HBOT COMMITTEE’S ACCEPTED INDICATIONS LIST


BREACH OF THE PULMONARY FILTER WITH TRANSESOPHAGEAL BUBBLE

ATTENUATION OF LIPID PEROXIDATION IN PORCINE CEREBRAL CORTEX AFTER A PROLONGED 25-MINUTE CARDIOPULMONARY ARREST BY HIGH-DOSE HYPERBARIC OXYGEN

ELECTRICAL DEFIBRILLATION TO TREAT VENTRICULAR FIBRILLATION IN A CONTROLLED, NON-RANDOMIZED PILOT IN SWINE





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