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Traumatic Brain Injury Treated with Low Pressure Hyperbaric Oxygen Therapy and SPECT Scans Show Progressive Medical Improvement

The brain injury program is a direct outgrowth of the extensive experience of Dr. Harch treating chronically brain injured patients, the independent 22 year clinical Hyperbaric Oxygen Therapy experience of Dr. Harch's practice group (Van Meter and Associates) at the Jo Ellen Smith Medical Center Hyperbaric Medicine Unit, the published reports of Drs. Richard A Neubauer and S.F. Gottlieb, and the 27 year clinical experience with brain injury of Dr. R.A Neubauer in Florida. In the late 1980's while at the Jo Ellen Smith Hyperbaric Medicine Unit, Dr. Harch observed patients with cerebral decompression sickness and/or air embolism who responded to hyperbaric oxygen therapy long after treatment of inert gas bubbles had passed or with delayed treatment months to years after standard U.S. Navy treatments had achieved partial success. In addition, Dr. Harch observed patients with brain contusion or stroke who, months to years after their neurological injury, concomitantly experienced gratuitous neurological improvement during the course of Hyperbaric Oxygen treatment for one of the standard accepted indications.

Harch HBOT in Cerebral Palsy and Pediatric Neurology: A Scientific Perspective

This article will review the current literature and history of application of low pressure (low dosage) LPHBOT to pediatric neurology. The most rigorous study on this subject will be analyzed and its interpretation debated in terms of past and present scientific data and theoretical considerations. The major flaw in the study's conclusion is illustrated by pre and post HBOT SPECT brain imaging on two of the author's cerebral palsy patients and the author's 12-year-experiehce of HBOT treatment of Cerebral Palsy children. There is substantial scientific explanation and data to argue for reimbursement of HBOT in Cerebral Palsy.

HBOT treatment for patients with Alcoholism, Drug Addiction, and Narcotic Addiction in Post-Intoxication & Abstinence Period

Hyperbaric Oxygenation in the Treatment of Patients with
Drug Addiction, Narcotic Addiction and Alcoholism
 in the Post-Intoxication and Abstinence Periods

[Original Article in Russian]
Epifanova NM.

H.R. 396: The Traumatic Brain Injury Treatment Act Has Been Added to the House Armed Services Bill

H.R. 396: The Traumatic Brain Injury Treatment Act Has Been Added to the House Armed Services Bill

On May 25, 2011, at 7:22 PM, William Duncan wrote:
Ok. Now that H.R. 396: The Traumatic Brain Injury Treatment Act has been added to the House Armed Services bill today by Congressman Sessions I have had several people ask me what that means and what the process is. Keep in mind it has typically taken 3 years to pass legislation in my other legislative projects. Persistence is key. This is year 3.

Harch Hyperbarics and The Family Physicians’ Center Congratulate Our HBOT Lottery Winners

Harch Hyperbarics and The Family Physicians’ Center
Congratulate Our HBOT Lottery Winners




Shany, Lara, and Tyler

To all of the lovely people who have written in expressing their desire to try this life changing therapy, we would like to say:
  • Never give up hope
  • You are on the right track by researching Hyperbaric Oxygen Therapy (HBOT). We urge you to continue to learn about HBOT, and write to your congressmen and senators. Stress them to advocate for insurance reimbursement for hyperbaric oxygen therapy for neurological disorders and traumatic brain injuries (TBI). 
  • If you did not win, we still have your story and you will be entered in the next lottery for another chance to win.
At Harch Hyperbarics and Family Physicians' Center we believe in doing everything we can to help the patients.

Hyperbaric Oxygen Therapy in Emergency Medicine

HBOT in Emergency Medicine

K. Van Meter, L. Weiss, and P.G. Harch


As an entry from K. K. Jain's Textbook Of Hyperbaric Medicine all chapter references refer to the 4th Edition.
Hypoxemia and ischemia are the underlying pathologies in many of the conditions seen in an emergency department. In addition to resuscitation and other emergency treatments, hyperbaric oxygen plays a vital role in the management of these patients. This topic is discussed under the following headings:
Timely resuscitation by augmentation of oxygen delivery to tissue damaged by ischemia is key to many emergency medicine interventions in sickness and injury. Further, the prompt attempt to lessen reperfusion injury and necrosis after initial resuscitative clinical success should not be forgotten. Finally, the patient, once past the initial resuscitative effort, followed by restorative oxygenation, should receive maintenance oxygenation as needed to optimize the chance of continued recovery. In other words, one of the major purposes of an emergency department is to first assure proper oxygen delivery to many of its sick and injured. The oxygen delivery must be adjusted to maximize therapeutic effect in the emergency medicine interventional phases of resuscitation and restoration, and the maintenance phase of patient management.
Oxygen delivery to tissue is dependent on cardiac output. Oxygen delivery to tissue is given by the following formula:
Oxygen delivery = cardiac output x arterial oxygen content 
(Shannon &Celli 1991)

Hyperbaric Oxygen Therapy in the Management of Cerebral Palsy

HBOT in the Management of Cerebral Palsy

Virginia Neubauer, Richard Neubauer and Paul Harch


An entry from K.K. Jain's Textbook Of Hyperbaric Medicine

Cerebral palsy is a chronic neurological disorder that can be due to several causes of brain damage in utero, in the perinatal period, or postnatally. Hyperbaric oxygen has been shown to be useful in treating children with cerebral palsy. This topic is discussed under following headings:

Causes of Cerebral Palsy Oxygen Therapy in the Neonatal Period Treatment of Cerebral Palsy with HBOT Conclusions


Causes of Cerebral Palsy

The term cerebral palsy (CP) covers a group of non-progressive, but often changing, motor impairment syndromes secondary to lesions or anomalies of the brain arising in the early stages of development. Between 20 to 25 of every 10,000 live-born children in the Western world have the condition (Stanley et al 2000). Problems may occur in utero, perinatal, and postnatal. Infections, traumatic brain injury, near-drowning and strokes in children suffering from neurological problems come under the heading of cerebral palsy. Diagnosis of cerebral palsy resulting from in utero or early perinatal causes may be made immediately after birth, but more commonly occurs between 15 and 24 months. It is possible that CP may be misdiagnosed for years because specific symptoms may show up very late in childhood. Some of the possible causes of Cerebral Palsy and are listed in Table 21.1.
Although several antepartum causes have been described for CP, the role of intrapartum asphyxia in neonatal encephalopathy and seizures in term infants is not clear. There is no evidence that brain damage occurs before birth. A study using brain MRI or post-mortem examination was conducted in 351 full-term infants with neonatal encephalopathy, early seizures, or both to distinguish between lesions acquired antenatally and those that developed in the intrapartum and early postpartum period (Cowan et al 2003). Infants with major congenital malformations or obvious chromosomal disorders were excluded. Brain images showed evidence of an acute insult without established injury or atrophy in (80%) of infants with neonatal encephalopathy and evidence of perinatal asphyxia. Although the results cannot exclude the possibility that antenatal or genetic factors might predispose some infants to perinatal brain injury, the data strongly suggest that events in the immediate perinatal period are most important in neonatal brain injury. These findings are important from management point of view as HBOT therapy in the perinatal period may be of value in preventing the evolution of cerebral palsy.

We Welcome Our Patients to Harch Hyperbarics and the Family Physicians' Center

Harch Hyperbarics and
The Family Physicians' Center


Approximately 90% of our patients fly in from out of town.

We do what we can to assist in making arrangements for their comfort while they are here in New Orleans. 
The need has arisen to create a SERVICE where certified Nurses’ Aides would be available to assist in the care of out of town patients.
Our Nurses’ Aides are available to help take care of your loved one in a hospital setting, Long Term Care (LTC) Facility, Rehab, or Ronald McDonald House.
 Our Nurses’ Aides work closely with an RN who mentors them and is available on call 24 hours a day, 7 days a week.

For more information, just ask Juliette at the private office.

(504) 309-4948
Veterans Call Toll Free 1-(855)-438-4268




Hyperbaric Oxygen Theraphy in Global Cerebral Ischemia/Anoxia and Coma

HBOT Therapy in Global Cerebral Ischemia Anoxia and Coma

Paul G. Harch and Richard A. Neubauer

 An entry from K.K. Jain's Textbook Of Hyperbaric Medicine

Hyperbaric oxygen therapy has been used in a number of conditions characterized by global ischemia (as opposed to focal ischemia of stroke), and anoxia, and leading to impairment of consciousness. Conditions such as coma due to brain injury and anoxia associated with drowning and hanging are discussed under the following headings:



For a discussion of the effectiveness of hyperbaric oxygen (HBOT) therapy in global cerebral ischemia/anoxia and coma, we define HBOT as a medical treatment that uses high pressure oxygen as a drug by fully enclosing a person or animal in a pressure vessel and then adjusting the dose of the drug to treat pathophysiologic processes of the diseases. Like all drugs, the dose of HBOT is crucial and should be customized to each patient's response. It is dictated by the pathological target and is determined by the pressure of oxygen, duration of exposure, frequency, total number of treatments, and timing of the dose in the course of the disease. As diseases and their pathologies evolve, different doses of HBOT are required at different times. In addition, patients have individual susceptibility to drugs, manifest side effects and toxicity. Unfortunately, the ideal dose of HBOT in acute or chronic global ischemia/anoxia and coma is unknown. The studies reviewed below suggest higher pressures (2 ATA or higher) and lesser numbers of treatments very early in the disease process whereas lower pressures (2 ATA or lower) and a greater number of treatments have been used as the brain injury matures. While this general trend seems justified, the absolute or effective pressures delivered to the patients in these reports may be slightly less than what is stated since many studies do not specify the HBOT delivery system that was employed. For example, an oxygen pressurized chamber has an effective HBOT pressure equal to the plateau pressure administered during the treatment, whereas an air pressurized chamber in which oxygen is administered by aviators mask can achieve a far lower effective HBOT pressure, depending on the fit of the mask and the amount of its air/oxygen leak. In the later cases, the dose of oxygen is less. This concept is particularly important when analyzing the studies in this chapter performed prior to the late 1980s when the aviator mask dominated delivery systems in multiplace chambers.


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